| Number of patients (% of total) |
Results |
Interpretation |
| 115 (63.5) |
Normal |
Normal Function of Gastric Mucosa |
| 36 (19.9) |
Helicobacter pylori Positive |
Non-atrophic risk of gastritis, related to H. pylori infection. Increased risk of peptic ulcer disease (duodenal or gastric).
-Treatment of H. pylori infection is recommended if successful therapy has not been given to the patient earlier. |
| 13 (7.2) |
Raised Pepsinogen |
Pepsinogen levels in the blood reflect the structure and function of the gastric corpus mucosa. A high PGI may indicate high acid output or ongoing PPI-medication. |
| 8 (4.4) |
All analytes raised |
Non-atrophic risk of gastritis, related to H.pylori infection.
Increased risk of peptic ulcer disease (duodenal or gastric).
-Treatment of H.pylori infection is recommended if successful therapy has not been given
to the patient earlier.
-High PGI value may indicate high acid output and/or ongoing PPI medication. |
| 5 (2.7) |
Raised Pepsinogen and Gastrin-17 (G17) |
A high PGI may indicate high acid output or ongoing PPI-medication. The blood G17 level reflects the structure and function of the mucosa in gastric antrum. The fasting blood level of G17 falls when the acidity of the stomach increases (pH < 2.5). Fasting G17 level < 1 pmol/L indicates very high acid secretion is very high and atrophy of the antrum mucosa, with loss of antral G cells. Fasting level of G17 >10 pmol/L, usually indicates a hypoacidic stomach due to PPI medication, or limited to corpus mucosa atrophy. |
| 4 (2.2) |
Low Pepsinogen (<30 ug/L) |
PGI levels < 30 ug/L indicate atrophy of the corpus mucosa |