| Combined |
Models of epistasis† |
| genotypes* |
1.1 |
1.2 |
2.1 |
2.2 |
3.1 |
3.2 |
4 |
5 |
6 |
7 |
8 |
| AA/BB |
0 |
0 |
0 |
0 |
0 |
0 |
0 |
0 |
0 |
0 |
0 |
| AA/Bb |
1 |
2 |
0 |
0 |
0 |
0 |
1 |
0 |
1 |
0 |
1 |
| AA/bb |
1 |
2 |
1 |
2 |
0 |
0 |
1 |
1 |
1 |
1 |
2 |
| Aa/BB |
2 |
1 |
0 |
0 |
0 |
0 |
1 |
0 |
1 |
0 |
1 |
| Aa/Bb |
2 |
2 |
0 |
0 |
0 |
0 |
1 |
0 |
2 |
0 |
2 |
| Aa/bb |
2 |
2 |
1 |
2 |
1 |
0 |
1 |
1 |
2 |
1 |
3 |
| aa/BB |
2 |
1 |
2 |
1 |
0 |
0 |
1 |
1 |
1 |
1 |
2 |
| aa/Bb |
2 |
2 |
2 |
1 |
0 |
1 |
1 |
1 |
2 |
1 |
3 |
| aa/bb |
2 |
2 |
2 |
2 |
1 |
1 |
1 |
1 |
2 |
2 |
4 |
*A, B: wild type alleles at loci A and B, respectively; a, b: variant alleles at loci A
and B, respectively; †1: Dominant epistasis; 2: Recessive epistasis; 3: Dominant
and recessive epistasis; 4: Double dominant epistasis without cumulative effect;
5: Double recessive epistasis without cumulative effects; 6: Double dominant
epistasis with cumulative effect; 7: Double recessive epistasis with cumulative
effects; 8: Quantitative; Of these, only the quantitative model is actually quantitative
(the numbers in the other models represent categories). Models that have two
versions (e.g., 1.1 and 1.2) are the cases where which SNP corresponds to the
“A” hypothetical locus and which corresponds to the “B” hypothetical locus is not
redundant. |
