Tendinitis is inflammation or irritation of a tendon, Tendons transmit the forces of muscle to the skeleton. As such, they are subjected to repeated mechanical loads, which are felt to be a major causative factor in the development of tendinopathy. Pathologic findings include tendon inflammation, mucoid degeneration, and fibrinoid necrosis in tendons. Microtearing and proliferation of fibroblasts have also been reported. However, the exact pathogenesis of tendinopathy is unclear.
Several research studies have made a significant contribution to different areas of tendon treatment. It is crucial to know how the current concepts on tendon mechanical behavior, surgery, and rehabilitation interact and may contribute to reach an optimal clinical outcome. The objective of this article is to review all factors that may affect the optimal length and tension restoration after AT rupture.