Characteristic   Healthy Subjects Patients Who Had Diffuse Axonal Injury
        Subacute P ≤ † Chronic P ≤ ‡
Peak frequency in central areas          
  EEG (Hz)            
    Left 13.9 ± 0.2 13.4 ± 0.1* .05 13.8 ± 0.2 .05
    Right 13.9 ± 0.2 13.5 ± 0.1* .05 13.9 ± 0.2 .05
  MEG (Hz)            
    Left 13.7 ± 0.2 13.3 ± 0.1* .05 13.7 ± 0.2 .05
    Right 13.7 ± 0.1 13.3 ± 0.1* .05 13.8 ± 0.2 .05
Amplitude in central areas          
  EEG (μV)            
    Left 29 ± 10 28 ± 10* .05 29 ± 9 .05
    Right 30 ± 9 29.5 ± 0.1* .05 31 ± 8 .05
  MEG(fT/cm)          
    Left 84 ± 29 68 ± 32* .05 87 ± 38 .05
    Right 90 ± 34 70 ± 31* .05 101 ± 18 .05
Cortical activation source strength on MEG          
  Precentral            
    Left 39 ± 19 22 ± 12* .05 37 ± 16 .05
    Right 39 ± 20 17 ± 16* .05 40 ± 20 .05
  Postcentral          
    Left 38 ± 21 20 ± 18* .05 38 ± 20 .05
    Right 35 ± 17 21 ± 19* .05 36 ± 18 .05
*N = 7 healthy subjects and 8 patients who had diffuse axonal injury. Data reported as mean ± SD. There were no differences in mean peak frequency of fast spindles, amplitude, and cortical activation source strength in precentral and postcentral regions between patients who had diffuse axonal injury (chronic stage) and healthy
participants. Abbreviations: EEG, electroencephalography; MEG, magnetoencephalography
Difference between healthy subjects and patients who had diffuse axonal injury (subacute stage).
Patients who had diffuse axonal injury; difference between subacute and chronic stages.
Table 4: Characteristics of fast spindles in healthy subjects and patients who had diffuse axonal injury after traumatic brain inury*