| Garlic pharmacological actions |
Health-promoting benefits |
| Inhibit cell division, induce apoptosis, block carcinogen activation, enhance DNA repair, induce detoxifying enzymes |
Anticarcinogenic/Antimutagentic |
| Inhibit microbiological growth as antibiotics |
Antimicrobial (antifungal, antiviral, antibacterial) |
| Scavenge oxidizing agents, induce SOD, GPx, GST, catalase |
Antioxidant |
| Increase proinflammatory cytokine release, stimulate natural killer cells |
Immuomodulatory |
| Inhibit enzymes in cholesterol and fatty acid synthesis |
Anti-hypolipidemic |
| Inhibit cholesterol synthesis, enhance cholesterol turnover |
Anti-hypocholesterolemic |
| Inhibit angiotensin II, induce NO and H2S, cause vasodilation |
Anti-hypertensive |
| Stimulate insulin production, interfere glucose absorption |
Anti-diabetic |
| Reduce trombosane formation, change platelet membrane |
Anti-thrombotic |
| Increase GSH levels by induction of GST |
Hepatoprotective |
* Derived partially from the publications by Cardelle-Cobas et al. (2010) as well as
Salman et al. (1999)