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Wendy Hwang is post doctoeral fellow at Lab of Dr. wen-Hwa Lee , Genomics research Centre. Taiwan, Collaborative projects 2003 - 2004 Participation in the research project PRIN 2004 ""Characterization of KRIT1 and its molecular interactions with RAP1 and ICAP1A: physiological functions and role in the pathogenesis of Cerebral Cavernous Malformations."" 2012 Participation PRIN 2010-2011” Sciences 'omics' as a tool for translational research in neuroncology” Scientific Societies Member of the Italian Association of Genetics and Molecular Biology (AIBG) Member of the Italian Society of Neuroscience (SINS) Member of the Italian Association of Neuropathology and Clinical Neurobiology (AINPeNC) Member of the Federation of European Neurosciences Societies (FENS) Member of the International Society of Neuropathology (ISN) Collaborating Partners EPIRARE (European Platform for Rare Diseases Registries).She is referee for Journal of Neurology, Neurosurgery, and Psychiatry Journal of the Neurological Sciences International Journal of Medical Sciences She is author or co-author of several international scientific papers in the field of molecular genetics. Currently she plays her teaching activity in the Messina School of Medicine and in other courses, in some Graduate Schools and she is a member of the School Committee of two PhD. Messina 26th March, 2014
Cell biology, molecular genetics and population genetics, functional analysis of SNPs. Actually the main research field of Rosalia D’Angelo is related to molecular and genetics features of Cerebral Cavernous Malformations that occur in both sporadic form that familial, identifying new variants in the three CCM genes. In this context, is a member of a Network multidisciplinary research on cerebral cavernous malformations (CCM Italy www.ccmitalia.unito.it) that, with an integrated approach to research, it is aims to relate the clinical, genetic, histological, molecular and cellular pathology features of this disease with a view to a more rapid advancement of knowledge on the pathogenic mechanisms.