Horner syndrome results from an interruption of the sympathetic nerve supply to the eye and is characterized by the classic triad of miosis partial ptosis, and loss of hemifacial sweating.
Decreased sweating on the affected side of the face, Drooping eyelid (ptosis), Sinking of the eyeball into the face, Small (constricted) pupil (the black part in the center of the eye)
Treatment depends on the cause of the problem. There is no treatment for Horner syndrome itself. When certain drugs have caused for the symptoms of the condition, we need to consult our doctor and report for the unusuality. Avoid neck injury, so to avoid the hazards of acquiring Horner’s syndrome.Have or practice a healthy diet and lifestyle. By this we can avoid the predisposing factors of the disease condition
A 67-year-old man underwent magnetic resonance imaging (MRI) for occasional headaches. He had high blood pressure and hypercholesterolemia, was obese, and had previously smoked cigarettes, and had been treated with perindopril, amlodipine, low-dose acetylsalicylic acid, and simvastatin. MRI revealed chronic ischemic injuries in the left superficial middle cerebral artery (MCA) territory, associated with an extremely spindly left ICA, which was misinterpreted as ICA thrombosis . The left anterior cerebral artery and MCA were supplied by a large posterior communicating artery from the basilar artery