University of Texas, USA
University of Texas
George Perry is dean of the College of Sciences and professor of biology at The University of Texas at San Antonio. Perry is recognized in the field of Alzheimers disease research particularly for his work on oxidative stress. Perry received his bachelor of arts degree in zoology with high honorsfrom University of California, Santa Barbara. After graduation, he headed to Scripps Institution of Oceanography and obtained his Ph.D. in marine biology in 1979. He then received a postdoctoral fellowship in the Department of Cell Biology at Baylor College of Medicine. In 1982, Perry joined the faculty of Case Western Reserve University until he moved to UTSA in 2006. He is distinguished as one of the top Alzheimers disease researchers with over 900 publications, one of the top 100 most-cited scientists in neuroscience and behavior and one of the top 25 scientists in free radical research. Perry has been cited over 40,000 times (H=101) and is recognized as an ISI Highly Cited researcher. Perry is editor for numerous journals and is editor-in-chief for the Journal of Alzheimers Disease. He is a Fellow of the American Association for the Advancement of Sciences, Microscopy Society of America,Royal Society of Medicine, Royal Society of Chemistry, Society of Biology and the Linnean Society and past-president of the American Association of Neuropathologists.He has been elected member ofthe Spanish Royal Academy of Sciences, Mexican Academy of Sciences and Academy of Sciences Lisbon; Iberoamerican Molecular Biology Organization, Dana Alliance for Brain Initiatives and won the Distinguished Professional Mentor Award from the Society for the Advancement of Chicanos and Native Americans in Science, Senior Investigator Award from the International College of Geriatric psychoneuropharmacology,Senior Fulbright Fellow and the National Honor Plaque of Panama for exceptional contributions to Neuroscience. Perry is on Board of the National Organization of Portuguese Americans and serves as Director of Education and Research.
Research is primarily focused on the mechanism of formation and physiological consequences of the cytopathology of Alzheimer disease. He has played a key role in elucidating oxidative damage as the initial cytopathological abnormality in Alzheimer disease. He is currently working to determine the sequence of events leading to neuronal oxidative damage and the source of the increased oxygen radicals. His current studies focus on two issues: (i) the metabolic basis for the mitochondrial damage restricted to vulnerable neurons; and (ii) the consequences of RNA oxidation on protein synthesis rate and fidelity.