Alzheimer’s Disease Pathogenesis and Mechanisms

Beta amyloid plaques and Neurofibrillary tangles of tau protein Pathology are prime suspects of neuronal cell death in Alzheimer’s it caused by accumulation of abnormally folded A-beta and tau proteins  and Plaques of small peptides 39–43 amino acids to  form Beta-amyloid which comprises amyloid precursor protein (APP) and a transmembrane protein that pierce the neuron's membrane. It later forms a fibrils of amyloid protein clump which get deposited in neurons to form a dense senile plaques, amyloid fibrils disrupts the cell’s calcium ions homeostasis and induces apoptosis of neurons which results in neurodegeneration affecting brain functioning.

 

  • Pathobiology of ApoE
  • Pathogenesis of Alzheimers Disease
  • Cognitive Impairment and Neuroinflammation in Alzheimers Disease
  • Rehabilitation Therapies for Alzheimers Disease
  • Tau Pathology of Alzheimers Disease
  • Pathogenesis Of Diabetic Nephropathy
  • Synaptic And Neuritic Alterations
  • Rapid Progression
  • Cerebrovascular Pathology
  • Cerebral Amyloid Angiopathy
  • Cholinergic Hypothesis
  • Cellular Signaling and cell to cell transmission
  • Oxidative damage and Mitochondrial dysfunction
  • Blood-brain barrier
  • Stem cells and Cell death
  • Cellular signalling, kinases, phosphatases, calcium
  • Neural networks, plasticity

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