Cancer Gene Therapy

Cancer therapies are drugs or other substances that block the growth and spread of cancer by interfering with specific molecules ("molecular targets") that are involved in the growth, progression, and spread of cancer. Many cancer therapies have been approved by the Food and Drug Administration (FDA) to treat specific types of cancer. The development of targeted therapies requires the identification of good targets that is, targets that play a key role in cancer cell growth and survival. One approach to identify potential targets is to compare the amounts of individual proteins in cancer cells with those in normal cells.  Proteins that are present in cancer cells but not normal cells or that are more abundant in cancer cells would be potential targets, especially if they are known to be involved in cell growth or survival.

Related Conferences: International Conference on Cervical Cancer, 22-23 September, 2016 (Vienna, Austria); 6th World Congress on Cancer Therapy, 01-03 December, 2016 (Baltimore, USA); 13th Global Summit on Cancer Therapy 17-19 October, 2016 (Dubai, UAE); International Conference on Pancreatic and Colorectal Cancer, 29-30 March, 2016 (Atlanta, USA); Global Summit on Melanoma And Carcinoma, 14-15 July, 2016 (Brisbane, Australia); Chromatin and Epigenetic in Cancer ( Atlanta, Georgia); Advances in Ovarian Cancer Research: Exploiting Vulnerabilities (Orlando, Florida); Advance in Breast Cancer Research ( Washington, DC); Advances in Pediatric Cancer Research (Florida);  New Horizons in Cancer Research Conference (Sanghai, China).

Cancer occurrs by the production of multiple mutations in a single cell that causes it to proliferate out of control. Cancer cells often different from their normal neighbors by a host of specific phenotypic changes, such as rapid division rate, invasion of new cellular territories, high metabolic rate, and altered shape. Some of those mutations may be transmitted from the parents through the germ line. Others arise de novo in the somatic cell lineage of a particular cell. Cancer-promoting mutations can be identified in a variety of ways. They can be cloned and studied to learn how they can be controlled. T lymphocytes have the capacity to hone in on tumor tissue. This property has been used to deliver cytokines directly to tumor masses for human gene therapy. The secretion of cytokines locally at the tumor site by the effector T lymphocytes will enhance their anti-tumor activity and avoid the side-effects that result from the systemic administration of cytokines.

  • Gene therapy in cancer
  • Breast cancer gene therapy
  • Lung cancer gene therapy
  • Prostate cancer gene therapy
  • Pancreatic cancer gene therapy
  • Characteristics of cancer cells
  • Cell cycle & oncology
  • Modern approaches in molecular cancer therapeutics
  • Genomic & cancer drug resistance
  • Genetically engineered cancer vaccines
  • Therapeutics cancer vaccines
  • Challenges & risks of cancer gene therapy

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Cancer Gene Therapy Conference Speakers

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