Paediatric Infectobesity

In addition to the imbalance between energy intake and expenditure, sedentary lifestyle, a diet high in saturated fats and sugars, and genetic predisposition, many other factors may be involved in obesity. The presence of either symbiotic or pathogenic microorganisms may contribute to the development of obesity. Gut microorganisms are believed to be involved in the development of obesity by two different and complementary mechanisms. They can extract energy from non-digestible polysaccharides and produce low-grade inflammation. Although the primary cause of obesity will always be a misbalance between energy intake and expenditure, its association with microbiology is only a small demonstration of how complex this multifactorial condition is. Nevertheless, beneficial microorganisms as well as pathogenic microorganisms deserve more attention as evidence of their contribution to metabolic disorders continues to accumulate.


Most studies done to date on adults have found a connection between exposure to Ad-36 and obesity, and all studies done so far on childhood obesity show an increase in prevalence of infection in obese children compared to non-obese children. The Ad-36 virus appears to both increase the number of fat cells by mobilizing precursor stem cells and increase the accumulation of fat within the cells. 

  • Role of Adenovirus SMAM-1 & Adenovirus-36 (Ad-36)
  • Food Intolerance & weight gain connection
  • Interactions Between the Gut Microbiota and Host Metabolism
  • Roles of gut bacteria in diabetes, obesity driven by genetic factors

Related Conference of Paediatric Infectobesity

October 25-26, 2018

21st World Obesity Conference

Budapest, Hungary
November 29-30, 2018

International Conference on Obesity and Diet Imbalance

Bali, Indonesia
June 21-22, 2019

21st Global Obesity Meeting

Brisbane, Australia

Paediatric Infectobesity Conference Speakers

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