Horner syndrome results from an interruption of the sympathetic nerve supply to the eye and is characterized by the classic triad of miosis partial ptosis, and loss of hemifacial sweating.
Decreased sweating on the affected side of the face, Drooping eyelid (ptosis), Sinking of the eyeball into the face, Small (constricted) pupil (the black part in the center of the eye)
Treatment depends on the cause of the problem. There is no treatment for Horner syndrome itself. When certain drugs have caused for the symptoms of the condition, we need to consult our doctor and report for the unusuality. Avoid neck injury, so to avoid the hazards of acquiring Horner’s syndrome.Have or practice a healthy diet and lifestyle. By this we can avoid the predisposing factors of the disease condition
Topical Cocaine may be used to confirm Horner's syndrome in subtle cases. Cocaine blocks reuptake of the neurotransmitter norepinephrine from the synaptic cleft and will cause dilation of the pupil with intact sympathetic innervation. One hour after instillation of two drops of 10% cocaine, the normal pupil dilates more than the Horner's pupil, thus increasing the degree of anisocoria. It is becoming increasingly difficult to obtain cocaine eye drops due to increased regulations.Topical Apraclonidine is an alternative to topical cocaine to confirm Horner's syndrome. Apraclonidine is an alpha adrenergic agonist. It causes pupillary dilation in the Horner's pupil due to denervation supersensitivity while producing a mild pupillary constriction in the normal pupil presumably by down-regulating the norepinephrine release at the synaptic cleft. A reversal of anisocoria after instilling two drops of 0.5% apraclonidine is suggestive of Horner's syndrome.