alexa Heart attack | Israel| PDF | PPT| Case Reports | Symptoms | Treatment

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Heart Attack

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  • Heart attack

    Pathology

    A myocardial infraction or a heart attack is permanent damage to the heart muscle. It occurs when the flow of oxygen-rich blood to heart muscle gets suddenly blocked by which the heart can’t get oxygen. Various symptoms include pain or discomfort in one or both the arms that spread to shoulders, upper back, neck or jaw, a general sense of anxiety, shortness of breath, stomach or abdominal pain, weakness and overwhelming fatigue, swelling of the ankles and lower legs, etc.

  • Heart attack

    Treatment

    A heart attack is a medical emergency so should call the ambulance. If the causality is not allergic to aspirin and its easily available than give them a tablet ideally 300mg to slowly chew and swallow while waiting for the ambulance to arrive. The aspirin will help to reduce the risk of heart attack by thinning the blood. The two main treatments include: using medication to dissolve blood clots, surgery to restore blood to the heart.

  • Heart attack

    Statistics

    In 2008 there were 39, 241 deaths in Israel, which represents half a percent (0.5%) of Israel's population in that year. Of these, 49.7% were male and 50.3% were female. Seventy-nine percent of the deceased were aged 65 and over (63% aged 75 and over and 32% aged 85 and over), 7% were under age 45, and 1.5% (605) were infants under one year of age. As shown in Table 1, the ten most common causes of death in 2008 were responsible for 77% of the total number of deaths in that year.

Expert PPTs

Speaker PPTs

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    Classically, the 3’untranslated region (3’UTR) is that region in eukaryotic protein-coding genes from the translation termination codon to the polyA signal. It is transcribed as an integral part of the mRNA encoded by the gene. However, there exists another kind of RNA, which consists of the 3’UTR alone, without all other elements in mRNA such as 5’UTR and coding region. The importance of independent 3’UTR RNA (referred as I3’UTR) was prompted by results of artificially introducing such RNA species into malignant mammalian cells. Since 1991, we found that the middle part of the 3’UTR of the human nuclear factor for interleukin-6 (NF-IL6) or C/EBP gene exerted tumor suppression effect in vivo. Our subsequent studies showed that transfection of C/EBP 3’UTR led to down-regulation of several genes favorable for malignancy and to up-regulation of some genes favorable for phenotypic reversion. Also, it was shown that the sequences near the termini of the C/EBP 3’UTR were important for its tumor suppression activity. Then, the C/EBP 3’UTR was found to directly inhibit the phosphorylation activity of protein kinase CPKC in SMMC-7721, a hepatocarcinoma cell line. Recently, an AU-rich region in the C/EBP 3’UTR was found also to be responsible for its tumor suppression. Recently we have also found evidence that the independent C/EBP 3’UTR RNA is actually exists in human tissues, such as fetal liver and heart, pregnant uterus, senescent fibroblasts etc. Through 1990’s to 2000’s, world scientists found several 3’UTR RNAs that functioned as artificial independent RNAs in cancer cells and resulted in tumor suppression. Interestingly, majority of genes for these RNAs have promoter-like structures in their 3’UTR regions, although the existence of their transcribed products as independent 3’UTR RNAs is still to be confirmed. Our studies indicate that the independent 3’UTR RNA is a novel non-coding RNA species whose function should be the regulation not of the expression of their original mRNA, but of some essential life activities of the cell as a whole.
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