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Horner syndrome results from an interruption of the sympathetic nerve supply to the eye and is characterized by the classic triad of miosis partial ptosis, and loss of hemifacial sweating.
Decreased sweating on the affected side of the face, Drooping eyelid (ptosis), Sinking of the eyeball into the face, Small (constricted) pupil (the black part in the center of the eye)
Treatment depends on the cause of the problem. There is no treatment for Horner syndrome itself. When certain drugs have caused for the symptoms of the condition, we need to consult our doctor and report for the unusuality. Avoid neck injury, so to avoid the hazards of acquiring Horner’s syndrome.Have or practice a healthy diet and lifestyle. By this we can avoid the predisposing factors of the disease condition
A 25-year-old man was admitted to our hospital for ophthalmologic surgical treatment of right ptosis. According to the patient's mother, the patient was delivered with the aid of forceps at birth, and the right ptosis was observed during the first few days of his life. At 2 to 3 years of age, his parents noted lighter color of the right eye. The right ptosis was gradually improved as he grew older. However, he developed right ptosis again with left meralgia paresthesia since eighteen age. At age 25 years, he was noted to have right ptosis, right miosis (the left pupil measured 4.5 mm in diameter and the right 3.0 mm), right heterochromia iridis with pigmented iris nevi, and left meralgia paresthesia . Laboratory data of urine, blood and CSF as well as radiological studies of chest X-ray, skull X-ray, spine X-ray, brain MRI and spinal cord MRI showed unremarkable. Sweating test was intact, pharmacologic test to Horner's syndrome with 5% cocaine and 1.25% 1-epinephrine indicated that the damage was pointed to the post ganglionic sympathetic neuron. Ten patients with congenital Horner's syndrome reported in Japan since 1953 were reviewed including our case.