Horner syndrome results from an interruption of the sympathetic nerve supply to the eye and is characterized by the classic triad of miosis partial ptosis, and loss of hemifacial sweating.
Decreased sweating on the affected side of the face, Drooping eyelid (ptosis), Sinking of the eyeball into the face, Small (constricted) pupil (the black part in the center of the eye)
Treatment depends on the cause of the problem. There is no treatment for Horner syndrome itself. When certain drugs have caused for the symptoms of the condition, we need to consult our doctor and report for the unusuality. Avoid neck injury, so to avoid the hazards of acquiring Horner’s syndrome.Have or practice a healthy diet and lifestyle. By this we can avoid the predisposing factors of the disease condition
Based on localization of the nerve pathway interruption, a Horner's syndrome is often classified as central, pre- or postganglionic. For the central type the syndrome is associated with other symptoms and signs from the central nervous system. The preganglionic type is most often caused by a tumor or trauma. The postganglionic type is often associated with pain/headache; most frequently it is seen as a consequence of carotid artery dissection or during cluster headache. Anhidrosis is rarely prominent, and in the postganglionic subtype it is virtually absent. Pharmacological tests can be used in diagnostics. Apraclonidine seems to be a good alternative to cocaine to confirm Horner's syndrome. MRI is generally recommended in the evaluation, if necessary with special sequences.