alexa Is Gut Microbiota Good to Acute Myocardial Infarction | OMICS International
ISSN: 2572-9462
Journal of Thrombosis and Circulation: Open Access

Like us on:

Make the best use of Scientific Research and information from our 700+ peer reviewed, Open Access Journals that operates with the help of 50,000+ Editorial Board Members and esteemed reviewers and 1000+ Scientific associations in Medical, Clinical, Pharmaceutical, Engineering, Technology and Management Fields.
Meet Inspiring Speakers and Experts at our 3000+ Global Conferenceseries Events with over 600+ Conferences, 1200+ Symposiums and 1200+ Workshops on
Medical, Pharma, Engineering, Science, Technology and Business

Is Gut Microbiota Good to Acute Myocardial Infarction

Chunling Kong* and Hongxing Luo

Department of Cardiology, Zhengzhou University People’s hospital, Zhengzhou, Henan, China

Corresponding Author:
Chunling Kong
Department of Cardiology
Zhengzhou University People’s hospital
Zhengzhou, Henan, China
Tel: 86-15515917818
E-mail: [email protected]

Received Date: August 14, 2017; Accepted Date: August 17, 2017; Published Date: August 24, 2017

Citation: Kong C, Luo H (2017) Is Gut Microbiota Good to Acute Myocardial Infarction. J Thrombo Cir 3:e109. doi: 10.4172/2572-9462.1000e109

Copyright: © 2017 Kong C, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Visit for more related articles at Journal of Thrombosis and Circulation: Open Access


The gut is home to hundreds and millions of bacteria that we call intestinal microbiota. They absorb nutrients from the human body and eliminate the metabolites to the body [1]. There may be some balance between gut microbiota and acute myocardial infarction so that we human can keep healthy.

Acute myocardial infarction (MI) refers to acute myocardial ischemic necrosis, a disease characterized on the basis of coronary artery atherosclerosis occurs more sharply reduce coronary blood supply or interrupt, leading to irreversible damage. According to the china cardiovascular disease report in 2016, urban residents with myocardial infarction mortality up to 70 per 100 thousands [2]. Recently, gut microbiota is given more attention on acute myocardial infarction.

Current research mainly focuses on the relation between gut microbiota and acute myocardial infarction. It proves that kinds and number of gut microbiota change with human health condition; some animal experiments indicate that the metabolic product of intestinal microbiota, such as TMAO has been proved to be an independent risk factor for coronary heart disease and we can reduce the mortality of acute myocardial infarction by regulating intestinal microbiota [3]. Some studies have shown that there are significant changes in the structure of intestinal microbiota, the helpful bacteria decreased significantly and harmful bacteria significantly increased, further aggravating the disease for the patient with acute myocardial infarction.

It is believed that the intestinal tract and the bacteria that live in it together form the endocrine organs. Intestinal microbiota affects normal physiological function by converting nutrients into hormone signals. Some intestinal microbial products can be perceived by human receptors and thus affect the progression of cardiovascular disease [4]. The mechanism of intestinal bacteria affecting acute myocardial infarction is still unclear and further study is needed.

However, studies on gut microbiota and acute myocardial infarction are still at animal experimental stage, not yet in clinical practice. We cannot make a conclusion whether gut microbiota is good or bad and the answer is still to be researched.

There was a significant difference on intestinal microbiota between healthy people and patients with acute myocardial infarction. Under normal circumstances, intestinal flora for healthy people relatively fixed, but when people are in acute myocardial infarction (MI), intestinal barrier function is impaired by a lack of heart pump function and intestinal perfusion inadequacy. Such as intestinal flora disorder, the intestinal mucosa and intestinal microbiota disorder further aggravates intestinal function damage, leading to bacterial endotoxin hematic disease, systemic inflammatory response and multiple organ failure, further damage to heart muscle cells. Therefore, intestinal flora structure is essential for the prognosis of acute myocardial infarction.

The causal relationship between intestinal flora and acute myocardial infarction is not yet determined; but the disorder of intestinal flora structure aggravated acute myocardial infarction and poor prognosis; maintaining the structure of intestinal flora may reduce the incidence of acute myocardial infarction, which is desirable and promising. More studies need to be done.


Select your language of interest to view the total content in your interested language
Post your comment

Share This Article

Recommended Journals

Recommended Conferences

  • International Conference on Medical and Health Science
    August 24-25, 2018 Tokyo, Japan
  • 11th International Conference on Clinical and Medical Case Reports
    October 22-23, 2018 Istanbul, Turkey
  • 3rd International Conference on Integrative Medicine and Alternative treatments
    October 22-23, 2018 Boston, USA

Article Usage

  • Total views: 301
  • [From(publication date):
    September-2017 - Jul 17, 2018]
  • Breakdown by view type
  • HTML page views : 270
  • PDF downloads : 31

Post your comment

captcha   Reload  Can't read the image? click here to refresh

Peer Reviewed Journals
Make the best use of Scientific Research and information from our 700 + peer reviewed, Open Access Journals
International Conferences 2018-19
Meet Inspiring Speakers and Experts at our 3000+ Global Annual Meetings

Contact Us

Agri & Aquaculture Journals

Dr. Krish

[email protected]

+1-702-714-7001Extn: 9040

Biochemistry Journals

Datta A

[email protected]

1-702-714-7001Extn: 9037

Business & Management Journals


[email protected]

1-702-714-7001Extn: 9042

Chemistry Journals

Gabriel Shaw

[email protected]

1-702-714-7001Extn: 9040

Clinical Journals

Datta A

[email protected]

1-702-714-7001Extn: 9037

Engineering Journals

James Franklin

[email protected]

1-702-714-7001Extn: 9042

Food & Nutrition Journals

Katie Wilson

[email protected]

1-702-714-7001Extn: 9042

General Science

Andrea Jason

[email protected]

1-702-714-7001Extn: 9043

Genetics & Molecular Biology Journals

Anna Melissa

[email protected]

1-702-714-7001Extn: 9006

Immunology & Microbiology Journals

David Gorantl

[email protected]

1-702-714-7001Extn: 9014

Materials Science Journals

Rachle Green

[email protected]

1-702-714-7001Extn: 9039

Nursing & Health Care Journals

Stephanie Skinner

[email protected]

1-702-714-7001Extn: 9039

Medical Journals

Nimmi Anna

[email protected]

1-702-714-7001Extn: 9038

Neuroscience & Psychology Journals

Nathan T

[email protected]

1-702-714-7001Extn: 9041

Pharmaceutical Sciences Journals

Ann Jose

[email protected]

1-702-714-7001Extn: 9007

Social & Political Science Journals

Steve Harry

[email protected]

1-702-714-7001Extn: 9042

© 2008- 2018 OMICS International - Open Access Publisher. Best viewed in Mozilla Firefox | Google Chrome | Above IE 7.0 version
Leave Your Message 24x7