alexa Acute Atrial Fibrillation Developed by the Inhalation o
ISSN: 2161-0495

Journal of Clinical Toxicology
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Case Report

Acute Atrial Fibrillation Developed by the Inhalation of Chlorine Gas

nYuksel Dogan1*, Aliye Soylu2, Sebahat N. Dogan3, Alpaslan Sahin1, Gulay A. Eren4 and Osman Karakaya1

1Department of Cardiology, Bakirkoy Dr. Sadi Konuk Education and Research Hospital, Istanbul, Turkey

2Department of Gastroenterology, Bakirkoy Dr. Sadi Konuk Education and Research Hospital, Istanbul, Turkey

3Department of Radiology, Taksim Education and Research Hospital, Istanbul, Turkey

4Department of Anesthesiology and Intensive Care, Bakirkoy Dr. Sadi Konuk Education and Research Hospital, Istanbul, Turkey

*Corresponding Author:
Yuksel Dogan M.D
Bakirkoy Dr. Sadi Konuk Education and Research Hospital
Cardiology Department, Istanbul, Turkey
Tel: +90 212 414 75 11
Fax: +90 212 583 77 57
E-mail: [email protected]

Received Date: November 08, 2011; Accepted Date: December 17, 2011; Published Date: December 19, 2011

Citation: Dogan Y, Soylu A, Dogan SN, Sahin A, Eren GA, et al. (2011) Acute Atrial Fibrillation Developed by the Inhalation of Chlorine Gas. J Clinic Toxicol 1:113. doi: 10.4172/2161-0495.1000113

Copyright: © 2011 Dogan Y, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

 

Abstract

Inhalation of chlorine gas is reported to cause upper and lower respiratory tract irritation, bronchospasm, respiratory failure and tachycardia. 31-year-old male patient was exposed to hydrochloric acid and chlorine bleach inhalation for 20 minutes. 48 hours after exposure, he was admitted to the emergency room with the complaints of increasing dyspnea and palpitation. The patient was tachypneic and tachycardic. His electrocardiogram revealed an atrial fibrillation at a rate of 145/min. The hemodynamics of the patient were stable, his hemogram, cardiac enzymes and other biochemical parameter measurements were normal. The patient was diagnosed with acute atrial fibrillation, he did not have any organic cardiac disease, as he was unresponsive to the administration of parenteral propafenone and metoprolol, he was treated with amiodarone infusion. 13 hours after the administration of amiodarone, his heart converted back to sinus rhythm. His control electrocardiogram and echocardiogram did not reveal any other pathology. He was discharged with metoprolol maintenance treatment.

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