alexa Advances in the Management of Valvular Heart Disease | OMICS International
ISSN: 2155-9880

Journal of Clinical & Experimental Cardiology
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Review Article

Advances in the Management of Valvular Heart Disease

Peter A. McCullough* and Krittapoom Akrawinthawong
St. John Providence Health System, Warren, MI, Providence Hospitals and Medical Centers, Southfield and Novi, MI, St. John Hospital and Medical Center, Detroit, MI, USA
Corresponding Author : Peter A. McCullough, MD
MPH, FACC, FACP, FAHA, FCCP, Consultant Cardiologist
Chief Academic and Scientific Officer
St. John Providence Health System
Providence Park Heart Institute
47601 Grand River Avenue, Suite B-125 Novi, MI 48374, USA
Tel: 248-465-5485
Fax: 248-465-5486
E-mail: [email protected]
Received: August 27, 2012; Accepted: August 27, 2012; Published: August 28, 2012
Citation: McCullough PA, Akrawinthawong K (2012) Advances in the Management of Valvular Heart Disease. J Clin Exp Cardiolog S3:e001. doi:10.4172/2155-9880.S3-e001
Copyright: © 2012 McCullough PA, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited
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Abstract

In recent decades, there has been considerable progress in the diagnosis and management of valvular heart disease. Use of echocardiography has been a major advance in understanding the pathophysiological aspects of common valvular diseases. In this issue of the Journal, Lacerda and Orton described a model to investigate the effects of chronic tensile stress on the mitral valve with inferences to those with myxomatous degeneration. Here, the important points are that chronic tensile loading puts undo mechanical forces on vulnerable parts of the valve and clearly contributes to progressive degenerative changes resulting in regurgitation. This model also adds on the evidences of robust relationship between mechanical load, pathologic mechanosensing pathway and structural change occurring at cellular level of valve [1-4]. Promising intervention to stop such vicious cycle at particular point is expectable to prevent or slow progression of disease.

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