alexa Auditory Hallucinations and Its Mechanism
ISSN: 2329-6895

Journal of Neurological Disorders
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Review Article

Auditory Hallucinations and Its Mechanism

Mizuno T*
Mikokoro Clinic, Nishi tenma 5-14-2, Kita-ku, Osaka, 530-0047 and Insutitut de la Philosophie Psychiatrique des serviteurs de la Notre-Dame, Kitashirakawa higashiogura cho 23-3, Sakyo-ku, Kyoto, 606-8265, Japan
Corresponding Author : Tatsuo Mizuno
Mikokoro Clinic, Nishi tenma 5-14- 2, Kita-ku, Osaka
530-0047 and Insutitut de la Philosophie
Psychiatrique des serviteurs de la Notre-Dame
Kitashirakawa higashiogura cho 23-3
Sakyo-ku, Kyoto, 606-8265, Japan
Tel: 81663611230
E-mail: [email protected]
Received: May 08, 2015; Accepted: May 13, 2015; Published:May 16, 2015
Citation: Mizuno T (2015) Auditory Hallucinations and Its Mechanism. J Neurol Disord 3:230. doi: 10.4172/2329-6895.1000230
Copyright: © 2015 Mizuno T. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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Abstract

Auditory hallucinations are one of the most frequent and reliable symptoms of psychosis. However, the neurocognitive and neurophysiological bases of auditory and verbal hallucinations remain obscure. These hallucinations are thought to result from disordered monitoring of inner speech. In this article, the synaptic and psychopathological mechanisms for the auditory hallucinations are discussed. Auditory hallucinations might be occurred due to a failure of synaptic connectivity. The disturbances of the spine are caused by temporary abnormal neuronal excitations and failure of synaptic connectivity will be observed. The abnormality in the neurotransmission of glutamic acid and GABA will cause a musical hallucinations, heightened auditory awareness and “Les eidolies hallucinosiques” (which are hallucinations without delusions). By adding the abnormal neurotransmission of dopamine and /or serotonin to GABA and glutamate abnormalities, the delusions will be applied to the “Les eidolies hallucinosiques” and it will become paranoid hallucinations (“Les hallucinations délirantes”). The valproic acid that inhibits the GABA degrading enzyme and agonist of the NMDA receptor will be the first choice for “Les eidolies hallucinosiques” from immediately after onset. If the patient complains the paranoid symptoms, the dopamine and serotonin receptor antagonist will be desirable.

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