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Bilateral Cerebellar Infarcts from Vertebral Artery Insufficiency Caused by Cervical Osteophytes | OMICS International | Abstract
ISSN: 2165-7939

Journal of Spine
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Case Report

Bilateral Cerebellar Infarcts from Vertebral Artery Insufficiency Caused by Cervical Osteophytes

Ripul R Panchal*, Daniel S Hutton and Kee D Kim
Department of Neurological Surgery, University of California-Davis Medical Center, Sacramento, California, USA
Corresponding Author : Ripul R Panchal
Department of Neurological Surgery
University of California
Davis School of Medicine
4860 Y Street, Suite 3740
Sacramento, CA 95817, USA
Tel: 916-734-3658
Fax: 916-452-2580
E-mail: [email protected]
Received June 20, 2012; Accepted July 25, 2012; Published July 27, 2012
Citation: Panchal RR, Hutton DS, Kim KD (2012) Bilateral Cerebellar Infarcts from Vertebral Artery Insufficiency Caused by Cervical Osteophytes. J Spine 1:122. doi:10.4172/2165-7939.1000122
Copyright: © 2012 Panchal RR, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Abstract

Background: In previous reports, the patients are described to have transient symptoms from physiologic rotation or extension of the cervical spine, resulting from a cervical osteophyte compressing the vertebral artery and causing vertebral artery insufficiency, known as Bow Hunter syndrome.

Methods: An 85-year-old female presented with new onset occipital headaches, nausea, vomiting and vertigo that were not precipitated by change in head position. Patient had bilateral cerebellar infracts. Patient underwent decompression and instrumented stabilization of the cervical spine from the posterior approach.

Results: At one-year follow-up, patient remained stroke free with patent vertebral artery.

Conclusion: To our knowledge, this is the first report of bilateral infraction from a vertebral artery insufficiency caused by cervical osteophytes without history of transient symptoms from movement of the head or neck, a variant of the Bow Hunter syndrome. Anterior versus posterior approach for vertebral artery insufficiency from osteophytic compression should be primarily based on location of the pathology and not the cervical level of involvement.

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