Bilateral Rapidly Destructive Arthrosis Case Report and ReviewLidia Sanchez-Riera*, Jinna Jagan Reddy and Mustafa Al Maini
Center on Ageing and Mobility, Zurich, Outside America Switzerland
- *Corresponding Author:
- Lidia Sanchez-Riera
Center on Ageing and Mobility
Zurich, Outside America, Switzerland
E-mail: [email protected]
Received date: August 07 2015; Accepted date: November 02, 2015; Published date: November 04, 2015
Citation: Sanchez-Riera L, Reddy JJ, Al Maini M (2015) Bilateral Rapidly Destructive Arthrosis Case Report and Review. Rheumatology (Sunnyvale) 5:174. doi: 10.4172/2161-1149.1000174
Copyright: © 2015 Sanchez-Riera L, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Rapidly destructive osteoarthritis or rapidly destructive arthrosis (RDA) of the hip is an uncommon disorder of unknown etiology where there is a rapid destruction of both the acetabulum and the femoral head. The condition typically affects elderly females with normal or osteoarthritic hip joints, and presents with new-onset severe hip pain and dysfunction. It is thought to be caused by extremely rapid osteoarthritic changes leading to an impact of the femoral head into the acetabulum, with subsequent osteonecrosis and insufficiency fracture of the femoral head. Differential diagnosis should include those conditions known to potentially lead to rapid hip destruction, such as septic arthritis, metabolic bone diseases, autoimmune inflammatory arthritis, malignancy and classical osteonecrosis. Sequential X-rays in patients with fast worsening of hip symptoms and a high degree of clinical suspicion seem mandatory to avoid extensive joint destruction and facilitate better arthroplasty outcomes in these patients. In the present report we present a clinical case of bilateral RDA, and we offer a useful review for clinicians on the differential diagnosis of this condition and the main physiopathological mechanisms behind its occurrence.