alexa <p>Detection of Undiagnosed Wilson and#195;and#162;and#194;and#8364;and#194;and#8482; s Disease after Hepatitis A Virus Infection</p> | OMICS International | Abstract
ISSN: 2167-0889

Journal of Liver
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Case Report

Detection of Undiagnosed Wilson ’ s Disease after Hepatitis A Virus Infection

Reza Dabiri, Ali Bastani and Amir Houshang Mohammad Alizadeh*
Gastroenterology and Liver Disease Research Center, Shahid Beheshti University of Medical Sciences, Tehran, Iran
Corresponding Author : Mohammad Alizadeh AH
Shahid Beheshti University of Medical Sciences
Taleghani Hospital, Parvaneh Ave
Tabnak Str., Evin, Tehran
Iran-19857, P.O.Box: 19835-178
Tel: 0098-21-22432521
Fax: 0098-21-22432517
E-mail: [email protected]
Received September 21, 2013; Accepted October 18, 2013; Published October 26, 2013
Citation: Dabiri R, Bastani A, Alizadeh AHM (2013) Detection of Undiagnosed Wilson's Disease after Hepatitis A Virus Infection. J Liver 2:130. doi: 10.4172/2167-0889.1000130
Copyright: © 2013 Dabiri R, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Abstract

Introduction: Some reports have considered hepatitis A as a possible factor in the development of acute decompensation in patients with Wilson’s disease. Here we report the case of a delayed diagnosis of Wilson’s disease in the end of the third decade of life in a patient infected with hepatitis A. Case report: The patient was a 26 year old woman with complaints of nausea and vomiting, anorexia, ichter, fever and epigastric pain since a week before admission. She had not any history of previous disease in herself and her families. Considering an increase in liver enzymes, serum bilirubin levels, serum IgM HAV antibody positivity and symptoms suggestive of hepatitis A associated with autoimmune hepatitis considered and patient was treated and then discharged. The patient returned two weeks later and symptoms such as ichter, fatigue and edema of the lower extremities were still present. Wilson’s disease is suspected, laboratory testing and ophthalmologic examination was performed and diagnosis was confirmed. The patient was treated with D-penicillamin, pyridoxine, and zinc sulfate. On reexamination, the patient’s symptoms largely resolved and in the following experiments response to treatment was appropriate. Conclusion: Hepatitis A can be considered as a factor for acute decompensation in undiagnosed patients with Wilson’s disease.

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