alexa Esophageal Stenosis as a Cause of Spontaneous Esophageal Perforation (Boerhaave Syndrome): A Case Report and Explanation of Possible Mechanism
ISSN: 2167-1222

Journal of Trauma & Treatment
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Case Report

Esophageal Stenosis as a Cause of Spontaneous Esophageal Perforation (Boerhaave Syndrome): A Case Report and Explanation of Possible Mechanism

Necdet OZ1 and Ahmet Bulent Kargi2*

1Thoracic Surgery Department, Medstar Hospital, Antalya, Turkey

2Thoracic Surgery Department, Kemerburgaz University, Istanbul, Turkey

*Corresponding Author:
Ahmet Bulent Kargi
Kemerburgaz University, Thoracic Surgery Department
Istanbul, Turkey
Tel: +902423143183
Fax: +902423143030
Email: [email protected]l.com

Received Date: March 31, 2016; Accepted Date: July 13, 2016; Published Date: July 15, 2016

Citation: Necdet OZ, Kargi AB (2016) Esophageal Stenosis as a Cause of Spontaneous Esophageal Perforation (Boerhaave Syndrome): A Case Report and Explanation of Possible Mechanism. J Trauma Treat 5:319. doi: 10.4172/2167-1222.1000319

Copyright: © 2016 Necdet OZ, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

 

Abstract

Introduction: Boerhaave syndrome (BS) is a spontaneous perforation of the esophagus which depends on increased intra-esophageal pressure, while the upper sphincter is closed during excessive vomiting. Case: A 75-year-old man was referred to hospital with sudden chest pain after vomiting. A computed chest tomography demonstrated mediastinal emphysema, thickening of the wall at the esophago-gastric junction level, and left pleural effusion. An upper gastrointestinal endoscopy showed an esophageal stenosis at mid thoracic portion and a perforation detected just 1 cm above of the lower gastro-esophageal sphincter. The patient underwent left posterolateral thoracotomy in the 12th hour of event. Stenotic segment is dilated and the mucosal perforation was repaired. Conclusion: BS is a serious disease with high morbidity and mortality rates. While BS usually occurs in a normal esophagus; in our case, BS was due to esophageal benign stenosis instead of upper sphincter esophageal sphincter closure. Benign stenosis may facilitate perforation as seen in our patient due to increased intraluminal pressure following vomiting.

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