alexa Hemorrhagic Stroke: Clinical, Etiologic and Evolutive Aspects in Senegalese Children
ISSN: 2329-6895

Journal of Neurological Disorders
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Research Article

Hemorrhagic Stroke: Clinical, Etiologic and Evolutive Aspects in Senegalese Children

Marième Soda Diop-Sène, Cissé Ousmane*, Moustapha Ndiaye, El-Hadj Malick Diouf, El-Hadj Makhtar Ba, Ngor Side Diagne, Amadou Gallo Diop and Mouhamadou Mansour Ndiaye

Neurology Department, Fann Teaching Hospital, Fann Avenue, Sheikh Cheikh Anta Diop BP: 5035, Dakar, Ivory Coast

*Corresponding Author:
Cisse Ousmane
Neurology Department Fann Teaching Hospital
Fann Avenue, Sheikh Cheikh Anta Diop BP: 5035
Dakar, Ivory Coast
Tel: 00221 77 507.87.51
E-mail: [email protected]

Received date: August 26, 2016; Accepted date: September 20, 2016; Published date: September 25, 2016

Citation: Diop-Sène MS, Ousmane C, Ndiaye M, Diouf EM, Makhtar Ba MH, et al. (2016) Hemorrhagic Stroke: Clinical, Etiologic and Evolutive Aspects in Senegalese Children. J Neurol Disord 4: 300. doi:10.4172/2329-6895.1000300

Copyright: © 2016 Diop-Sène MS, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.



Introduction: Hemorrhagic Stroke (HS) are uncommon among children. They have an etiological specificity and different risk factors that are different from those of adults. Prognosis depends on the etiology. Our objective was to study the characteristics of children's HS in Senegal. Methodology: Descriptive retrospective study from July 2003 to February 2014. Results: 13 patients were collected, including 9 boys. The mean age was 9.85 years. The antecedents were: rheumatic heart disease (2 patients); congenital heart disease (1 patient); arterial hypertension systolic-diastolic untreated (1 patient); heterozygous sickle cell anemia (1 patient). No history for (8 patients - 61.53%). The beginning was sudden or rapidly progressive in all patients. Clinical manifestations were hemiplegia (53.84%), headache and vomiting (46.15%), consciousness disorders (30.76%), partial motor seizures (7.69%). The topography was supratentorial (84.61%). The causes were: arteriovenous malformations (3 patients); severe thrombocytopenia (1 patient); systolic-diastolic arterial hypertension (1 patient); myeloblastic leukemia (1 patient). No etiology (7 patients). 4 patients died during the acute phase. 1 patient was transferred to pediatric oncology. 1 patient was transferred abroad for embolization. The mean follow-up was 11.8 months. Outcome was favorable in patients with ad integrum restitution. 7 patients had hemiparesis type of neurological sequelae (4 patients), motor partial epilepsy (4 patients) and aphasia (1 patient). 1 patient developed hydrocephalus. No recurrence was noted. Conclusion: This study provides a better diagnostic and etiological approach for better management.


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