alexa Intracranial Hypertension in Behcet Disease: A Case Report
ISSN: 2165-7920

Journal of Clinical Case Reports
Open Access

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Case Report

Intracranial Hypertension in Behcet Disease: A Case Report

Figen Guney1*, Banu Bozkurt2 and Yahya Paksoy3
1Department of Neurology, Necmettin Erbakan University, Turkey
2Department of Ophthalmology, Selcuk University, Turkey
3Department of Radiology, Selcuk University, Turkey
*Corresponding Author : Figen Guney
Department of Neurology, Necmettin
Erbakan University, Turkey
Tel: +90 332 2236380
Fax: +90 332 3340492
E-mail: [email protected]
Received October 12, 2015; Accepted March 26, 2016; Published March 31, 2016
Citation: Guney F, Bozkurt B, Paksoy Y (2016) Intracranial Hypertension in Behcet Disease: A Case Report. J Clin Case Rep 6:748. doi:10.4172/2165-7920.1000748
Copyright: © 2016 Guney F, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
 

Abstract

Behcet disease (BD) is a chronic multisystem disorder characterized predominantly by recurrent episodes of occlusive vasculitis. According to the accumulated data, it is seen that intracranial hypertension generally develops secondary to the cerebral venous sinus thrombosis in BD. In the study, it was aimed in light of literature to discuss a case where intracranial hypertension occurred without venous sinus thrombosis. A 36-year-old female was evaluated for the symptoms of blurred vision in her left eye for the last 3 months and transient visual obscurations in her right eye beginning 3 days ago. It was found out that the patient had been diagnosed with BD one and a half months ago. Fundus examination revealed bilateral swollen optic discs. Lumbar puncture revealed an opening pressure of 360 mm H2O, with normal composition. The patient was diagnosed with intracranial hypertension developing secondary to BD according to modified Dandy criteria. Methylprednisolone 1000 mg intravenously (IV) for 5 days, followed by prednisolone 60 mg po, was administered. A significant improvement was observed in the complaints and papilledema of the patient. Inflammation could play a significant role in the intracranial hypertension developing without venous sinus thrombosis in BD. As a result, BD should be considered in the differential diagnosis in patients with intracranial hypertension especially in areas where the disease prevalence is high.

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