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ISSN: 2155-9899

Journal of Clinical & Cellular Immunology
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Review Article

Mechanism of Host Cell Death in Response to Bacterial Infections

Zakir Hossain and Md. Fakruddin*
Institute of Food Science and Technology (IFST), Bangladesh Council of Scientific and Industrial Research (BCSIR), Dhaka, Bangladesh
Corresponding Author : Md. Fakruddin
Scientific Officer, Industrial Microbiology Laboratory
Institute of Food Science and Technology (IFST)
Bangladesh Council of Scientific and Industrial Research (BCSIR), Dhaka, Bangladesh
Tel: +8801717684750
E-mail: [email protected]
Received: June 19, 2012; Accepted: September 17, 2012; Published: September 27, 2012
Citation: Hossain Z, Fakruddin Md (2012) Mechanism of Host Cell Death in Response to Bacterial Infections. J Clin Cell Immunol 3:128. doi:10.4172/2155-9899.1000128
Copyright: © 2012 Hossain Z, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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Abstract

Viral and microbial infections often elicit programmed cell death as part of the host defense system or as a component of the survival strategy of the pathogen. Pathogens have evolved an array of toxins and virulence factors to modulate host cell death pathways. By inducing host cell death, bacteria and viruses eliminate key immune cells and evade host defenses that can compromise their viability. Apoptosis, necrosis, and pyroptosis represent the three major programmed cell death modes during infection, and the choice of death mode depends on a variety of factors, including the nature of the pathogen, pathogen load, and the site of infection. Further insight into the complex relationship between hosts and pathogens will be gained by further elucidating the molecular mechanisms underlying necrosis and pyroptosis, as well as by characterizing new mechanisms by which microbes induce and evade apoptotic, necrotic, and pyroptotic cell death in their hosts.

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