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ISSN: 2168-9547

Molecular Biology: Open Access
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Review Article

Mitochondria as a Favourite Organelle for Invading Viruses

Latif Reshi1,2 and Jiann-Ruey Hong1,3*

1Lab of Molecular Virology and Biotechnology, Institute of Biotechnology, National Cheng Kung University, Tainan City 701, Taiwan

2Department of Life Sciences, College of Bioscience and Biotechnology, National Cheng Kung University, Tainan City 701, Taiwan

3Department of Biotechnology and Bioindustry, National Cheng Kung University, Tainan City 701, Taiwan

*Corresponding Author:
Dr. Jiann-Ruey Hong
Laboratory of Molecular Virology and Biotechnology
Institute of Biotechnology, National Cheng Kung University
Tainan 701, Taiwan
Tel: 886-6-2003082
Fax: 886-6-2766505
E-mail: [email protected]

Received date: November 29, 2016; Accepted date: January 07, 2017; Published date: January 14, 2017

Citation: Reshi L, Hong JR (2017) Mitochondria as a Favourite Organelle for Invading Viruses. Mol Biol 6:181. doi:10.4172/2168-9547.1000181

Copyright: © 2017 Reshi L, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Abstract

The self-destruction of cells infected with viruses undergoes the process of apoptosis generally to restrict infection and the spread of viral progeny. To avoid infection host has evolved interconnected complex defence network that comprises innate and acquired immune response. Mitochondria being considered as powerhouse of a cell is not limited to only energy production, but mitochondria perform various other functions in (disease, apoptosis and host innate immune system) which make them absolutely indispensable to the cell. This makes them a target of almost all the invading pathogens including viruses. Therefore being a multifunctional organelle, the viruses choose mitochondria as a favourite organelle as they can easily take control of the whole cell and make it to promote or block apoptosis as per their need.

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