Pulmonary Edema (Non-Cardiogenic) and Acute Respiratory Distress Syndrome Secondary to Amniotic Fluid Embolism Syndrome Following Preterm Delivery of Intrauterine Fetal Death: A Case ReportVandita K Patil1, Usha Varghese1, Kailas N Patil2*, Swayed Mahmud Ali Reza2, Jawaher Al Yazeedi3, Shaji Varghese3 and Ashwin Varghese4
- Corresponding Author:
- Kailas N Patil
Department of Anaesthesiology
Buraimi Regional Referral Hospital, Oman
Tel: +968- 25650855
E-mail: [email protected]
Received date: June 16, 2016; Accepted date: July 14, 2016; Published date: July 20, 2016
Citation: Patil VK, Varghese U, Patil KN, Ali Reza SM, Yazeedi JA, et al. (2016) Pulmonary Edema (Non-Cardiogenic) and Acute Respiratory Distress Syndrome Secondary to Amniotic Fluid Embolism Syndrome Following Preterm Delivery of Intrauterine Fetal Death: A Case Report. J Clin Case Rep 6:843. doi:10.4172/2165-7920.1000843
Copyright: © 2016 Patil VK, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
A 27 years old healthy female of twenty eight weeks pregnancy with history of low grade fever and dry cough for one day presented with intrauterine fetal death. Following spontaneous preterm delivery of the dead fetus, within three hours, patient developed irritable cough, dyspnea, tachypnea, restlessness and cyanosis. She was put on face mask with oxygen flow of ten litres/minute and was nebulized with Salbutamol in the delivery suite but gradually she developed desaturation of 76%. As the condition of patient was worsening patient was transferred to intensive care unit. In intensive care unit patient was intubated and put on ventilator immediately. X-ray chest was showing bilateral infiltrates and arterial blood gases was showing PaO2/FiO2 ratio of 55.6%. Pulmonary capillary wedge pressure was not checked as pulmonary catheterization is not practiced in our Intensive care unit. Cardiogenic component of pulmonary edema was ruled out indirectly by history, electrocardiogram, echocardiography, central venous pressure and X-ray chest (heart shadow). She was diagnosed as a case of severe acute respiratory distress syndrome (American European consensus conference guideline, 2012) and non-cardiogenic pulmonary edema due to amniotic fluid embolism syndrome. In course of management, maximum emphasis was given on lung protective ventilation and fluid, conservative strategy along with medical and other supportive management. On her eighth day on ventilator, she was extubated and on ninth day, she was shifted to maternity ward. On fourteenth day she was discharged from hospital. She came for follow up after one month of her discharge and was found to have no residual complication.