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Quinalphos Induced Alterations in the Levels of Ions and Whole Animal Oxygen Consumption of Freshwater Fish, <em>Cyprinus Carpio</em> (Linnaeus, 1758) | Abstract
ISSN: 2157-7579

Journal of Veterinary Science & Technology
Open Access

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Research Article

Quinalphos Induced Alterations in the Levels of Ions and Whole Animal Oxygen Consumption of Freshwater Fish, Cyprinus Carpio (Linnaeus, 1758)

Sameer Gopal Chebbi and Muniswamy David*

 

Karnatak University’s research laboratory, environmental and molecular toxicology division, Department of Zoology, Karnatak Science College, Dharwad-580001, Karnataka, India

*Corresponding Author:
Dr. Muniswamy David
Karnatak Science College
Staff Quarters No. D-1
Udaya Hostel road,Dharwad
Karnataka,India
Tel: +919845709815
E-mail: [email protected]

Received date: August 05, 2010; Accepted date: September 14, 2010; Published date: September 14, 2010

Citation: Chebbi SG, David M (2010) Quinalphos Induced Alterations in the Levels of Ions and Whole Animal Oxygen Consumption of Freshwater Fish, Cyprinus Carpio (Linnaeus, 1758). J Veterinar Sci Technol 1:102. doi:10.4172/2157-7579.1000102

Copyright: © 2010 Chebbi SG, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Abstract

The fi ngerlings of Cyprinus carpio were exposed to the lethal concentration (7.5 g/l) of quinalphos (Emulsi fi ed Concentrate 25 percent) for 1,2,3 and 4 days respectively to study the levels and whole animal oxygen consumption. Exposed fi sh showed a signi fi cant decrease in the whole animal oxygen consumption due to the respiratory distress as a consequence of the impairment of oxidative metabolism. The ionic content of Na + , K + and Ca 2+ also decreased signi fi cantly in gill, liver and muscle tissue of exposed fi sh, signifying the altered cellular proliferation and deranged Na + , K + and Ca 2+ ionic pump due to probable consequence of tissue damage.

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