Role of Blood-Let out Cupping Therapy in Angina and Angina Risk Management.
|Salwa AM Nasrat1, Salah M El-Sayed2 and Abdullah M Nasrat3*|
|1Department of Physical Therapy, Cardiac Surgery Academy, Cairo, Egypt|
|2Department of Biochemistry and Molecular Biology, Taibah University, Medina, Saudi Arabia|
|3Department of Surgery, Balghsoon Clinic, Jeddah, Saudi Arabia|
|Corresponding Author :||Abdullah M Nasrat
Department of Surgery, Balghsoon Clinic
Jeddah, P.O. Box 52611, Jeddah 21573, Saudi Arabia
E-mail: [email protected]
|Received April 04, 2015; Accepted April 27, 2015; Published April 29, 2015|
|Citation: Nasrat SAM, El-Sayed SM, Nasrat AM (2015) Role of Blood-Let out Cupping Therapy in Angina and Angina Risk Management. J Clin Case Rep 5:523. doi:10.4172/2165-7920.1000523|
|Copyright: © 2015 Nasrat SAM, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.|
The study aimed at demonstration of the effect of cupping therapy in ischemic myocardial conditions. Despite advances in treatment of myocardial infarction, the mortality rate is still high. In spite of the efficacy and safety of coronary stenting, the overall adverse cardiac events after stent insertion is also high; 8.1%. Accumulation of toxic mediators in the body is a documented fact. These substances can induce vascular spasm and other effects on vascular endothelium. Impairment of the vascular endothelial function is involved in the pathogenesis of wide variety of cardiovascular diseases and hence is considered a therapeutic target. Production of ischemic metabolites within the myocardium due to lack of proper perfusion constitutes a toxic element which can contribute to progression of the coronary heart disease. Elimination of these toxic elements from the targeted tissues and circulation is a challenge that would definitely help to correct an underlying micro-circulatory error. Withdrawal of these ischemic tissue metabolites, being interstitial, is only feasible via superficial scratching on the skin and suction by the traditional therapeutic cups; a maneuver which could lead to correction of an associated spastic ischemic situation at the microcapillary level. Three male patients aged between 61-65 years with history of two coronary stents insertion, have developed chest tightness few months after coronary stenting. They hesitated towards undergoing further coronary procedures. They were scheduled for basic cupping therapy on the upper back and front of the chest. All patients demonstrated dramatic symptomatic relief and documented clinical recovery. Cupping therapy is promising in relieving ischemic myocardial conditions via elimination of inflammatory mediators that compromise the microcirculation.