alexa SiNiTang – A Traditional Chinese Herbal Remedy Improves Cardiac Function Post-MI
ISSN: 2155-9880

Journal of Clinical & Experimental Cardiology
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Research Article

SiNiTang – A Traditional Chinese Herbal Remedy Improves Cardiac Function Post-MI

Wolfgang Dietl1, Karola Trescher1, Philip Hohensinner1, Felix Nagel1, Yan Ma2, Margarita Wolfsberger3, Roberto Plasenzotti4, Karin Hoffmann-Sommergruber2, Johann Wojta1, Michael Bauer1* and Bruno K. Podesser1
1Ludwig Boltzmann Cluster for Cardiovascular Research, Medical University Vienna, Vienna, Austria
2Institute for Pathophysiology, Medical University Vienna, Vienna, Austria
3University Clinics for Pediatrics and Adolescent Medicine, Medical University of Vienna, Vienna, Austria
4Core Unit for Biomedical Research, Medical University of Vienna, Vienna, Austria
Corresponding Author : Dr. Michael Bauer
Ludwig Boltzmann Cluster for Cardiovascular Research
c/o Core Unit for Biomedical Research, Medical University of Vienna
Waehringer Guertel 18-20/1Q, 1090 Vienna, Austria
Tel : +43140400-5223
Fax: +43140400-5229
Email: [email protected]
Received November 07, 2011; Accepted January 17, 2012; Published January 20, 2012
Citation: Dietl W, Trescher K, Hohensinner P, Nagel F, Ma Y, et al. (2012) SiNiTang – A Traditional Chinese Herbal Remedy Improves Cardiac Function Post-MI. J Clinic Experiment Cardiol S5:004. doi:10.4172/2155-9880.S5-004
Copyright: © 2012 Dietl W, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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Abstract

SiNiTang is a well known traditional Chinese herbal remedy. It is used to treat cold extremities, weak pulse and lethargy: Symptoms that correspond with myocardial infarction and heart failure in western medicine. To examine potential beneficial effects on post-MI remodeling, a modified SiNiTang formula was tested using a well established model of myocardial infarction in Rats. Application of SiNiTang significantly reduced left ventricular dilation and thus preserved left ventricular function. However, it did not affect the hypertrophic response. Exploratory histopathological examination revealed better vascularization of infarct and peri-infarct areas in SiNiTang treated animals compared to placebo. HUVEC cells were treated with SiNiTang showed increased proliferation and tube formation. SiNiTang attenuates ventricular dilation post-MI and increases neoangiogenesis.

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