alexa Superficial Siderosis Following Posterior Fossa Explora
ISSN: 2168-975X

Brain Disorders & Therapy
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Case Report

Superficial Siderosis Following Posterior Fossa Exploration

James Gauci*, Reuben Grech and Josanne Aquilina
Department of Neuroscience, Mater Dei Hospital, Msida, Malta
Corresponding Author : James Gauci
Department of Neuroscience
Mater Dei Hospital Msida, Malta
Tel: 00356 9929 7386
E-mail: [email protected]
Received: July 06, 2015 Accepted: September 06, 2015 Published: September 14, 2015
Citation: Gauci J, Grech R, Aquilina J (2015) Superficial Siderosis Following Posterior Fossa Exploration. Brain Disord Ther 4:184. doi:10.4172/2168- 975X.1000184
Copyright: © 2015 Gauci J, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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A 70 year old lady presented with symptoms and signs suggesting lesions in the vestibulocochlear nerve, the cerebellum and brainstem. Features on magnetic resonance imaging of the brain led to the diagnosis of superficial siderosis. This condition is a very rare neurodegenerative disorder characterised by deposition of haemosiderin in several areas of the nervous system. This occurs a result of recurrent bleeding in the subarachnoid space. Various sources of chronic bleeding have been implicated, including dural defects, neoplasms or arterio-venous malformations. Whilst presenting symptoms depend on the site of haemosiderin deposition, there is a predilection for the eight cranial nerve, the cerebellum and brainstem. The investigation of choice is magnetic resonance imaging of the brain, and linear hypointensity is pathognomonic. Early identification of this condition will obviate the need for further, extensive investigation of the patient’s symptoms. It will also enable the physician to search for an underlying, potentially treatable cause. Our patient gave a history of posterior fossa exploration half a century prior to her current presentation, suggesting the presence of a dural defect as the cause of this disorder.


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