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ISSN: 2155-9899

Journal of Clinical & Cellular Immunology
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Review Article

The Role of Fcγ Receptors in Myocardial Diseases and Atherosclerosis

Chiharu Kishimoto1* and Zuyi Yuan2
1Department of Cardiovascular Medicine, Graduate School of Medicine, Kyoto University, 54 Kawaracho, Shogoin, Sakyo-ku, Kyoto 606-8507, Japan
2First Affiliated Hospital of Medical School, Xi’an Jiaotong University, 277 Yanta West Road, Xi’an, Shaanxi 710061, China
Corresponding Author : Chiharu Kishimoto
Department of Cardiovascular Medicine
Graduate School of Medicine
Kyoto University, 54 Kawaracho
Shogoin, Sakyo-ku, Kyoto 606-8507, Japan
Tel: +81-75-751-3197
Fax: +81-75-751-4281
E-mail: [email protected]
Received September 26, 2012; Accepted October 29, 2012; Published November 05, 2012
Citation: Kishimoto C, Yuan Z (2012) The Role of Fcγ Receptors in Myocardial Diseases and Atherosclerosis. J Clin Cell Immunol S10:004. doi:10.4172/2155-9899.S10-004
Copyright: © 2012 Kishimoto C, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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Abstract

Myocardial diseases and atherosclerosis are widely considered to be an immune mediated process. Fcγ receptors (FcγRs) contribute to the regulation of immune and inflammatory responses and have been implicated in human cardiovascular lesions. Major cell types involved in the pathogenesis of the diseases express FcγRs and their ligands such as immune complexes and C-reactive protein have been shown to activate FcγRs signal pathway. This review summarizes recent significant progress addressing the various roles of FcγRs in the disease pathogenesis which comes from the studies of FcγRs deficient animal models, clinical investigations and in vitro molecular and cellular studies. These new findings help us appreciate the emerging role of FcγRs in cardiovascular diseases, and suggest FcγRs as a potential therapeutic target for the diseases.

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