The Unusual Cause of Dangerous Arrhythmias at the Young
|Medical Disciplines Department, Faculty of Medicine and Pharmacy, University of Oradea, Romania|
|*Corresponding Author :||Manuela Stoicescu
Medical Disciplines Department
Faculty of Medicine and Pharmacy
University of Oradea, Romania
Tel: +40 259 408 105
E-mail: [email protected]
|Received date: March 28, 2016; Accepted date: April 20, 2016; Published date: April 30, 2016|
|Citation: Stoicescu M (2016) The Unusual Cause of Dangerous Arrhythmias at the Young. J Clin Exp Cardiolog 7:438. doi:10.4172/2155-9880.1000438|
|Copyright: © 2016 Stoicescu M, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.|
Objectives: The main objectives of this clinical case presentation was to found the real cause of dangerous arrhythmias (frequents polymorphic premature ventricular bates and repeated passes of none sustained ventricular tachycardia) at a young patient.
Methods: I present the clinical case of a young woman patient 21 years old, who came at the consultation for irregular palpitations after increase physical effort–mountain climbing-her favorite sport activity. At the objective examination were found irregular heart bates HR=98 bates/min, without murmur heart or added sounds, BP=150/90 mmHg, normal vesicular breath sound on the lung. The EKG showed: sinus rhythm, HR=98 bates/minutes, many polymorphic premature ventricular bates and a short pass of a non sustained ventricular tachycardia (less than 30 seconds). The patient was monitored and followed antiarrhythmic therapy with xilin iv bolus 1 mg/kg body and after that double dose of xilin 1% i.v. in perfusion during the first 24 hours with disappears of the dangerous arrhythmias and after that remain on beta-blocker therapy- Carvedilol 6, 25 mg daily. In the first instance these dangerous arrhythmias were put in context of increase physical effort. The usual laboratory tests and the specific cardiac enzymes: Troponin I and CPKMB were in normal range and was excluded a heart attack. The levels of TSH, Free T3, FreeT4 were normal and the thyroid ultrasound-normal, excluding a possible thyroidal disease (hyperthyroidism- Basedow Graves disease or toxic thyroid adenoma and cardiothyreosis The level of electrolytes (potassium, magnesium, calcium, sodium, chloride) were normal so a dyselectrolytemia was excluded like a cause of these dangerous arrhythmias. Transthoracic echocardiography put in evidence unexpected a solid mass inside of the left ventricular cavity. The patient was referred to the Cardiovascular Surgery Department. The formation was removed from the left ventricular cavity and the result of the histopathological examination confirmed safe the diagnosis of ventricular myxoma.
Results: The atrial myxoma is common in medical literature, but rare. Ventricular myxoma is very uncommon, rare and sometimes can develop so dangerous arrhythmias like repeated passes of ventricular tachycardia and also polymorphic premature ventricular bates and can put in danger patent’s life because of risk of degeneration in ventricular fibrillation and cardiac arrest.
Conclusion: Sometimes, rare, an unknown ventricular myxoma could be the real cause of dangerous arrhythmias in the young and must to be removed.