alexa The autistic neuron: troubled translation?
Neurology

Neurology

Autism-Open Access

Author(s): Kelleher RJ rd, Bear MF

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Abstract Autism is a complex genetic disorder, but single-gene disorders with a high prevalence of autism offer insight into its pathogenesis. Recent evidence suggests that some molecular defects in autism may interfere with the mechanisms of synaptic protein synthesis. We propose that aberrant synaptic protein synthesis may represent one possible pathway leading to autistic phenotypes, including cognitive impairment and savant abilities. This article was published in Cell and referenced in Autism-Open Access

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