The most common electrophysiologic mechanisms leading to SCD are tachyarrhythmias such as ventricular fibrillation (VF) or ventricular tachycardia (VT). Interruption of tachyarrhythmias, using either an automatic external defibrillator (AED) or an implantable cardioverter defibrillator (ICD), has been shown to be an effective treatment for VF and VT. The implantable defibrillator has become the central therapeutic factor in the prevention and treatment of sudden cardiac death.
Patients with tachyarrhythmias, especially VT, carry the best overall prognosis among patients with sudden cardiac arrest (SCA).Norms established in the USSR in 1977, updated in Russia in 1996, were applied: under 30 mg/dl—no adverse health effects; 30–50 mg/dl—insignificant intoxication; 50–149 mg/dl—slight intoxication; 150–249 mg/dl—medium intoxication; 250–299 mg/dl—strong intoxication; 300–499 mg/dl—advanced and potentially lethal intoxication; and above 500 mg/dl—lethal.
CPR: Immediate cardiopulmonary resuscitation (CPR) is critical to treating sudden cardiac arrest. By maintaining a flow of oxygen-rich blood to the body's vital organs, CPR can provide a vital link until more advanced emergency care is available. Ongoing Research is being done at cardiac centres.