700 Journals and 15,000,000 Readers Each Journal is getting 25,000+ ReadersThis Readership is 10 times more when compared to other Subscription Journals (Source: Google Analytics)
Case Report Open Access
Oculomotor nerve palsy has been recognized as a localizing sign due to aneurysms along the internal carotid artery, resulting from direct pressure on the nerve. There were only two case reports of an anterior communicating artery (AcomA) aneurysm rupture followed by bilateral oculomotor nerve palsy. We experienced the rare finding of a patient with transient bilateral oculomotor nerve palsy unrelated to consciousness disturbance that started about 38 hours after subarachnoid hemorrhage (SAH) due to AcomA aneurysm rupture. Without raised intracranial pressure, bilateral mydriasis resolved spontaneously 22 hours later. A thick clot Fisher group 3 SAH occupying perimesencephalic, interhemispheric, and sylvian cisterns bilaterally,remained in CT scans afterthe operation. Reconstructed MRI data of the oculomotor nerve were converted to a curved multiplanar reconstruction (MPR) image suggested that subarachnoid blood occupying the perimesencephalic cisterns bilaterally induced compression on the oculomotor nerves directly, and the subsequent local edema caused transient bilateral palsy.
Oculomotor nerve palsy, Subarachnoid hemorrhage, Aneurysm, Anterior communicating artery, Pupilary dilation, Neurosurgery Clinics Management, Endoscopic Neurosurgery