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Left Ventricular hypertrophy is outlined as an increase in the mass of the left ventricle, which may also be secondary to an expand in wall thickness, and broaden in cavity measurement. Left ventricular hypertrophy more often than not develops steadily. Whilst ventricular hypertrophy happens naturally as a response to aerobic undertaking and strength coaching, it is most commonly known as a pathological response to cardiovascular disorder, or high blood stress.
Treatment for LVH focuses on the underlying the cause of the condition. Depending on the cause, Treatment may include medication or surgery. Doctor suggests the treatment for LVH such as regular exercise a low-sodium, low-fat diet and no smoking. They used to control the blood pressure by using some types of drugs like enalapril (Vasotec), lisinopril (Prinivil, Zestril) and captopril (Capoten), atenolol (Tenormin), carvedilol (Coreg), metoprolol (Toprol XL) and bisoprolol (Zebeta) and etc.
We evaluated 21 hypertensive patients with electrocardiographic LVH in the LIFE study and 8 normal controls using 3D MR tagging and echocardiography. Patients had higher MR LV mass than normals (116 ± 40 versus 63 ± 6 g/m2, P = 0.002). Neither echocardiographic fractional shortening (32 ± 6 versus 33% ± 3%), LVEF (63% versus 64%) or mean end-systolic stress (175 ± 27 versus 146 ± 28 g/cm2) were significantly different, yet global MWCS was decreased by both echocardiography (13.4 ± 2.8 versus 18.2% ± 1.5%, P < 0.001) and MR (16.8 ± 3.6 versus 21.6% ± 3.0%, P < 0.005).
