An administration of HMGB1 via intracerebroventricular, intratracheal, intraperitoneal and intraarticular routes induces marked inflammatory responses, and activates various innate immune cells.
|
| Figure 1: HMGB1-mediated proinflammatory responses at
various sites An administration of HMGB1 via intracerebroventricular, intratracheal, intraperitoneal and intraarticular routes induces marked inflammatory responses, and activates various innate immune cells. |
