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Malignant Glaucoma

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Malignant Glaucoma

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It refers to sustained ongoing process that is difficult to treat and characteristically progresses to blindness. It is sometimes unresponsive and occasionally worsened with conventional management. The underlying pathology of malignant glaucoma is believed to be aqueous misdirection into or posterior to the vitreous body with anterior displacement of the lens-iris diaphragm.

Related Journals of Malignant Glaucoma

Journal of Glaucoma: Open Access, Optometry: Open Access, Journal of Clinical & Experimental Ophthalmology, International Journal of Ophthalmic Pathology, Journal of Current Glaucoma Practice, Journal of Glaucoma, Current Eye Research, Current Opinion in ophthalmology, American Journal of Ophthalmology, Progress in Retinal and Eye Research, Glia, Ophthalmology, Ophthalmologica, Japanese Journal of Ophthalmology, Journal Français d'Ophtalmologie, Journal of Cataract and Refractive Surgery, Ophthalmic Research, Survey of Ophthalmology.

Malignant Glaucoma

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  • Yosef Yarden
    Classically, the 3’untranslated region (3’UTR) is that region in eukaryotic protein-coding genes from the translation termination codon to the polyA signal. It is transcribed as an integral part of the mRNA encoded by the gene. However, there exists another kind of RNA, which consists of the 3’UTR alone, without all other elements in mRNA such as 5’UTR and coding region. The importance of independent 3’UTR RNA (referred as I3’UTR) was prompted by results of artificially introducing such RNA species into malignant mammalian cells. Since 1991, we found that the middle part of the 3’UTR of the human nuclear factor for interleukin-6 (NF-IL6) or C/EBP gene exerted tumor suppression effect in vivo. Our subsequent studies showed that transfection of C/EBP 3’UTR led to down-regulation of several genes favorable for malignancy and to up-regulation of some genes favorable for phenotypic reversion. Also, it was shown that the sequences near the termini of the C/EBP 3’UTR were important for its tumor suppression activity. Then, the C/EBP 3’UTR was found to directly inhibit the phosphorylation activity of protein kinase CPKC in SMMC-7721, a hepatocarcinoma cell line. Recently, an AU-rich region in the C/EBP 3’UTR was found also to be responsible for its tumor suppression. Recently we have also found evidence that the independent C/EBP 3’UTR RNA is actually exists in human tissues, such as fetal liver and heart, pregnant uterus, senescent fibroblasts etc. Through 1990’s to 2000’s, world scientists found several 3’UTR RNAs that functioned as artificial independent RNAs in cancer cells and resulted in tumor suppression. Interestingly, majority of genes for these RNAs have promoter-like structures in their 3’UTR regions, although the existence of their transcribed products as independent 3’UTR RNAs is still to be confirmed. Our studies indicate that the independent 3’UTR RNA is a novel non-coding RNA species whose function should be the regulation not of the expression of their original mRNA, but of some essential life activities of the cell as a whole.
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  • Richard A. Kenley
    Equilibrium binding interactions between lotrafilcon a soft contact lenses and the two prostaglandin anti-glaucoma drugs Bimatoprost and Tafluprost
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  • Lourdes Cortes-Dericks
    ALDHhigh/CD44+ putative cancer stem cell population as therapeutic target in malignant pleural mesothelioma
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  • Janardan Kumar
    An overview of RGD-containing peptides research and it’s potentials to the field of glaucoma therapy and/or vitreolysis
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