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Membranous Nephropathy

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  • Membranous nephropathy


    Membranous nephropathy
     (MEM-bruh-nus nuh-FROP-uh-thee) occurs when the small blood vessels in the kidney (glomeruli), which filter wastes from the blood, become inflamed and thickened. As a result, proteins leak from the damaged blood vessels into the urine (proteinuria).

  • Membranous nephropathy

    Disease pathophysiology:

    Loss of these proteins eventually causes signs and symptoms known as nephrotic syndrome. his glomerulopathy has the histological characteristic of lacking significant hypercellularity, and the electron microscopy shows subepithelial/intramembranous immune deposits (immunoglobulin G and complement), which cause podocyte damage and usually nephrotic syndrome.

  • Membranous nephropathy

    Disease statistics:

    For many, loss of these proteins eventually causes signs and symptoms known as nephrotic syndrome. his glomerulopathy has the histological characteristic of lacking significant hypercellularity, and the electron microscopy shows subepithelial/intramembranous immune deposits (immunoglobulin G and complement), which cause podocyte damage and usually nephrotic syndrome.

  • Membranous nephropathy

    Disease treatment:

    "Patients with nephrotic syndrome should be managed according to standard practice. Fluid retention should be managed using oral or IV diuretic therapy alongside appropriate fluid restriction. Intravenous albumin may be necessary in severe cases and ultrafiltration or haemodialysis may be required in resistant cases with oliguria or renal failure."

  • Membranous nephropathy

    Major research on disease:

    PLA2R is a protein found in cells in the kidney, and is involved in the development of MN. The disease occurs when the immune system causes antibodies to attack the PLA2R protein which then results in the thickening of the capillary walls in the kidney filters and the resulting damage causes proteinuria (protein in the urine) and kidney disease. "

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