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Oxidative stress reflects an imbalance between the systemic manifestation of reactive oxygen species and a biological system's ability to readily detoxify the reactive intermediates or to repair the resulting damage. Liver disease is relatively common but often occurs in the absence of specific clinical signs. The liver has great powers of regeneration and more overt clinical signs associated with its failure do not appear until some 70-80% of the functional capacity is lost. Obscure signs of liver disease are therefore much more common than overt signs of liver failure. It has been demonstrated that various inflammatory cytokines, such as tumor necrosis factor TNF-Î±, interferon (IFN-Î³), and interleukin IL-1Î² produced during liver injury are involved in promoting tissue damage. However, innate immune cells are also the main source of IL-10, IL- 6, and certain prostaglandins, all of which have been shown to play a hepatoprotective role.Moreover it was stated by GarcÃa-Ruiz et al. that liver injury and altered metabolism in liver disease also may be generated by inflammation-promoting substances called cytokines. Oxidative stress and cytokines could be used as biomarkers of liver abscess syndrome in Dromedary camel (Camelus dromedaries). Further investigations and extra efforts should be made to confirm this in other species. If this could be achieved, it would yield a valuable tool to diagnose liver abscess syndrome and would open up new perspectives in research fields dealing with liver abscess. (El-Deeb WM, Fouda TA,Liver Abscess in Dromedary Camels (Camelusdromedaries): Oxidative Stress Biomarkers and Proinflammatory Cytokines)
Last date updated on November, 2020