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Adverse health consequences due to inappropriate environmental perinatal conditions may become evident during adulthood as a result of the metabolic plasticity of mammalian organisms [1-3]. Specifically, studies performed by us and others in a CD-1 mice model have shown that pups repeatedly subjected to a mild nociceptive stress during lactation (NS-mice) present overweight in adulthood, in addition to an increased amount of epididymal fat and altered levels of glucose, insulin, lipids, leptin, corticosterone and adrenocorticotrophic hormone
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