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s reviewed elsewhere [1], amyloids are amorphous deposits of proteins folded into stacked β-sheets in the form of long fibrils. Amyloids occur in high levels in certain human diseases and research has traditionally focused on how amyloid may cause disease. The Aβ peptide that forms the brain amyloid plaques which characterise Alzheimer's disease (AD) is one of the most intensively studied molecules in the body, yet is still very poorly understood.
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