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One etiology of disorder is lumbar facet joint pathology, which seems to be the primary pain generator in approximately 10–15% of chronic LBP patients. Facet-related pain may be generated by different mechanisms, including secretion of inflammatory mediators into the facet joint cartilage and synovial tissue; distention and stretch of joint capsule by joint fluids; compression of the exiting nerve root in the neural foramen or spinal recesses by facet articular hypertrophy; spasm of the paraspinal muscles in response to capsular irritation or nerve entrapment by calcified mamillo accessory ligament
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