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IN Drosophila, most of our knowledge of meiosis has come from a number of elegant genetic studies which have utilized structural and numerical rearrangements as well as the normal chromosomal constitution to gain insights into meiotic chromosome behavior. These studies, although they have given US a precise formal description of chromosome behavior during meiotic recombination and segregation, have shed little light onto the control of this behavior. One approach toward an understanding of the genetic control of meiosis in Drosophila melunogaster was suggested by SANDLER et al. (1968) who undertook the systematic isolation and characterization of mutants in which meiotic chromosome behavior was abnormal. The assumption upon which this approach is based is that the normal functions of genes governing meiotic processes can be inferred from the abnormal meiotic chromosome behavior caused by mutants in such genes.
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