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Endothelial dysfunction is a well established response to cardiovascular risk factors and precedes the development of atherosclerosis. Endothelial dysfunction is involved in lesion formation by the promotion of both the early and late mechanisms of atherosclerosis including up-regulation of adhesion molecules, increased chemokine secretion and leukocyte adherence, increased cell permeability, enhanced low-density lipoprotein oxidation, platelet activation, cytokine elaboration, and vascular smooth muscle cell proliferation and migration. Endothelial dysfunction is a term that covers diminished production/availability of nitric oxide and/or an imbalance in the relative contribution of endothelium-derived relaxing and contracting factors. Also, when cardiovascular risk factors are treated the endothelial dysfunction is reversed and it is an independent predictor of cardiac events.
Related journals of Endothelial Dysfunction
Journal of Autacoids and Hormones, Archives of Medicine, Journal of Diabetic Complications & Medicine, Journal of Clinical & Cellular Immunology, Epidemiology: Open Access, Clinical Depression, Clinical and Experimental Psychology, Journal of Health Education Research & Development, Molecular Biology, Lancet Neurology, The Alzheimer's and Dementia, Annals of Neurology, Brain; a journal of neurology, Acta Neuropathologica, Nature Reviews Neurology, Stroke, Neurology.