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Immune thrombocytopenia (ITP) comprises a heterogeneous group of disorders characterized by autoimmune-mediated platelet destruction and impairment of platelet production [1,2]. Autoimmune-mediated accelerated platelet destruction with subsequent clearance in the reticuloendothelial (RE) system can be reduced by RE phagocytosis-preventing agents, such as intravenous immunoglobulin (IVIG), intravenous anti-D, or corticosteroids, or may be permanently resolved by splenectomy.
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