Oxidative Stress in Homocysteinemia: The Prevention of the Therapy-Induced Peripheral NeuropathyMohamed Shehata*
University of Cologne, Deutz-Kalker Street 118, 50679, Cologne, Germany
- *Corresponding Author:
- Dr. Mohamed Shehata
Deutz-Kalker Str. 118, 50679, Cologne, Germany
E-mail: [email protected]
Received Date: January 23, 2017; Accepted Date: March 13, 2017; Published Date: March 22, 2017
Citation: Mohamed Shehata (2017) Oxidative Stress in Homocysteinemia: The Prevention of the Therapy-Induced Peripheral Neuropathy. J Clin Exp Neuroimmunol 2:111.
Copyright: © 2017 Shehata M. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Oxidative stress (OS) is an unhealthy condition that results from the imbalance between the production of reactive oxygen species (ROS) and the rate of their elimination through the anti-oxidant defence mechanisms. This condition is a part of the pathogenesis of many disease conditions. In this paper, the development of OS in homocysteinemia is highlighted, where some light is shed on the mechanism of the increased ROS production and the potential protective strategy in the clinical practice.