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Journal of Alzheimers Disease & Parkinsonism received 3123 citations as per Google Scholar report
Articles published in Journal of Alzheimers Disease & Parkinsonism have been cited by esteemed scholars and scientists all around the world. Journal of Alzheimers Disease & Parkinsonism has got h-index 25, which means every article in Journal of Alzheimers Disease & Parkinsonism has got 25 average citations.
Following are the list of articles that have cited the articles published in Journal of Alzheimers Disease & Parkinsonism.
| 2023 | 2022 | 2021 | 2020 | 2019 | 2018 | 2017 | 2016 | |
|---|---|---|---|---|---|---|---|---|
Year wise published articles |
36 | 33 | 60 | 30 | 45 | 44 | 107 | 89 |
Year wise citations received |
123 | 389 | 489 | 425 | 374 | 263 | 237 | 253 |
| Journal total citations count | 3123 |
| Journal impact factor | 2.04 |
| Journal 5 years impact factor | 3.14 |
| Journal cite score | 10.01 |
| Journal h-index | 25 |
| Journal h-index since 2018 | 22 |
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Nanotoxicity and Risk Assessment |
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Modulation of the microbiota-gut-brain axis by bioactive food, prebiotics, and probiotics decelerates the course of Alzheimer's disease |
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Bile Acids as Key Modulators of the Brain-Gut-Microbiota Axis in Alzheimers Disease |
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Probiotics Fermentation Technology, a Novel Kefir Product, Ameliorates Cognitive Impairment in Streptozotocin-Induced Sporadic Alzheimers Disease in Mice |
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Roles of Gut Microbiota in Pathogenesis of Alzheimers Disease and Therapeutic Effects of Chinese Medicine |
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Implications of the Human GutBrain and GutCancer Axes for Future Nanomedicine |
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Crosstalk between Gut and Brain in Alzheimers Disease: The Role of Gut Microbiota Modulation Strategies |
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Dysbiosis and Alzheimers Disease: A Role for Chronic Stress? |
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Alternative Anti-Infective / Anti-Inflammatory Therapeutic Options for Fighting Alzheimers Disease |
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The Microbiome as a Therapeutic Target for Multiple Sclerosis: Can Genetically Engineered Probiotics Treat the Disease? |
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The MicrobiotaGutBrain Axis and Alzheimers Disease: Neuroinflammation Is to Blame? |
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Facilitation of Gastrointestinal (GI) Tract Microbiome-Derived Lipopolysaccharide (LPS) Entry Into Human Neurons by Amyloid Beta-42 (A?42) Peptide |
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Cell surface amyloid proteins of microorganisms: structure, properties and significance in medicine |
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The role of the gut microbiome in mediating neurotoxic outcomes to PCB exposure |
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A Gut Feeling: The Importance of the Intestinal Microbiota in Psychiatric Disorders |
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Age-related cerebral small vessel disease and inflammaging |
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Functional amyloids: interrelationship with other amyloids and therapeutic assessment to treat neurodegenerative diseases |
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Systemic Immune Dyshomeostasis Model and Pathways in Alzheimers Disease |
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Down-Regulation of Essential Synaptic Components by GI-Tract Microbiome-Derived Lipopolysaccharide (LPS) in LPS-Treated Human Neuronal-Glial (HNG) Cells in Primary Culture: Relevance to Alzheimers Disease (AD) |
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Accelerated Amyloid Beta Pathogenesis by Bacterial Amyloid FapC |
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